We are where we are with Afghanistan. And where is that, exactly?

Terrific blog post by Andrew Sullivan. Excerpt:

So how are we where are we now? In Afghanistan, the Taliban has been empowered by the long occupation and the government is as corrupt as ever and fast losing its own people. Al Qaeda have simply moved to Pakistan where they remain safely as long as they duck drone attacks. In Iraq, we actually gave al Qaeda a new opening and had to spend billions and lose thousands of lives to push them back. Even now, we have no guarantee they will not re-emerge in a still deeply divided country when far fewer American troops will soon remain. And through all this, we threw away one core advantage: our moral high ground. Through torture and the mass killings of civilians, through allowing sectarian genocide in Iraq and giving the world Abu Ghraib and Gitmo as symbols of the new America, we even managed to blur the lines between civilization and barbarism. And in this struggle, our political leaders failed to keep the country united, or the alliance intact.

The awful truth is: what 9/11 revealed, and what it was designed to reveal, is that there is nothing we can really do definitively to stop another one. They had no weapons but our own technology. The training they had was not that sophisticated and the costs of the operation were relatively tiny. There were 19 of them. None of the key perpetrators has been brought to justice. Bin Laden remains at large. If you calculate the costs of that evil attack against the financial, moral and human costs of the fight back, 9/11 was a fantastic demonstration of the power of asymmetry to destroy the West.

Everything that has subsequently transpired has merely deepened that lesson. The US is now bankrupt, trapped in Iraq and Afghanistan for the rest of our lives, unable even to prevent the two most potentially dangerous Islamist states, Pakistan and Iran, from getting nukes, morally compromised and hanging on to global support only because of a new president who is even now being assaulted viciously at home for such grievous crimes as trying to get more people access to health insurance…

Apple declares war…

… on unauthorised ingenuity, to wit this:

According to Wired,

the latest update to Snow Leopard, version 10.6.2, drops support for the Intel Atom processor. This means that anyone with a “hackintosh” who tries to update to the latest operating system version will see their computer die, going no further than the gray Apple logo on startup.

The reports are lighting up various hackintosh forums, and OSx86-co-author wizard Stellarolla sums it up thusly:

“Well, looks like I was right, again. The netbook forums are now blowing up with problems of 10.6.2 instant rebooting their Atom-based netbooks. My sources tell me that every time a netbook user installs 10.6.2 an Apple employee gets their wings.”

It shouldn’t be long before some clever hacker figures out a workaround and releases a patched kernel to the world, re-enabling the OS on Atom-based computers. But that’s not the story. The bigger message is that Apple has finally stopped ignoring the incessant buzz of the hobby-hacking, Mac netbook scene and instead pulled out a fly-swatter and dealt it a whack. The war is officially on.

Hee, hee! Thanks to a warning from Quentin, I shan’t be ungrading for the time being. And my lovely little Dell/Apple hybrid will continue to be a delightful workhorse.

The thing that gave me most pleasure when I hacked the Dell in the first place was that I was simultaneously annoying two of the world’s great control freaks! Apple’s latest gambit only increases the pleasure.

Hardwired news

We are, as George Steiner used to say, “language animals”. Noam Chomsky argued that we are born with, somehow, a natural capacity for language. Now comes some indications of the genetic mechanisms that could be responsible for our great gift.

The first concrete evidence of a genetic link to the evolution of language in humans was published today in the journal Nature. Researchers led by UCLA neurogeneticist Daniel Geschwind have shown that two small differences between the human and chimpanzee versions of a protein called FOXP2 result in significant differences in the behavior of dozens of genes in human neurons.

FOXP2 is a protein known as a transcription factor; its role is to turn other genes off or on. Geschwind and his collaborators deleted the native gene for FOXP2 from a lab-grown line of human neurons. They then inserted either the gene for human or chimp FOXP2 into the cells and screened the cells to see which genes were being expressed, or actively producing proteins. The researchers identified dozens of genes that were expressed at either higher or lower levels depending on whether the cells were making human or chimp FOXP2. They verified these findings by examining gene expression patterns in post-mortem brain tissue from both chimps and humans who died of natural causes…